Field dossier · from the corpus
Apolipoprotein E
0predicted collisions
Where this field is predicted to collide
No open collisions on this field in the current run.
Most cited work
Alzheimer's disease
The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases
APOE4 exacerbates synapse loss and neurodegeneration in Alzheimer’s disease patient iPSC-derived cerebral organoids
ApoE-Isoform-Dependent SARS-CoV-2 Neurotropism and Cellular Response